A new study further implicates low levels of the amino acid glycine in development of nonalcoholic fatty liver disease, or NAFLD. It also suggests addressing this might hold the key to a future treatment for the disease.
“We’ve uncovered a new metabolic pathway and potential novel treatment,” says senior author Y. Eugene Chen, M.D., Ph.D., a professor of internal medicine and surgery, from the Michigan Medicine Frankel Cardiovascular Center. His team collaborated with researchers from the University of Michigan, Wayne State University and Technion-Israel Institute of Technology.
