New insight on the molecular mechanisms that allow malaria parasites to move and spread disease within their hosts has been published today in the open-access eLife journal.
The movement and infectivity of the parasite Plasmodium falciparum, and ultimately its ability to spread malaria among humans, rely on a large molecular complex called the glideosome.
The new findings provide a blueprint for the design of future antimalarial treatments that target both the glideosome motor and the elements that regulate it.